Publicaciones en las que colabora con Marina Lasa Benito (14)

2024

  1. Autophagy sustains mitochondrial respiration and determines resistance to BRAFV600E inhibition in thyroid carcinoma cells

    Autophagy, Vol. 20, Núm. 6, pp. 1383-1397

  2. Protein kinase D activity is a risk biomarker in prostate cancer that drives cell invasion by a Snail/ERK dependent mechanism

    Biochimica et Biophysica Acta - Molecular Basis of Disease, Vol. 1870, Núm. 1

2021

  1. Downregulation of snail by dusp1 impairs cell migration and invasion through the inactivation of jnk and erk and is useful as a predictive factor in the prognosis of prostate cancer

    Cancers, Vol. 13, Núm. 5, pp. 1-21

  2. V600e braf inhibition induces cytoprotective autophagy through ampk in thyroid cancer cells

    International Journal of Molecular Sciences, Vol. 22, Núm. 11

2019

  1. Resveratrol promotes apoptosis through the induction of dual specificity phosphatase 1 and sensitizes prostate cancer cells to cisplatin

    Food and Chemical Toxicology, Vol. 124, pp. 273-279

2016

  1. TGFβ induces epithelial-mesenchymal transition of thyroid cancer cells by both the BRAF/MEK/ERK and Src/FAK pathways

    Molecular Carcinogenesis, Vol. 55, Núm. 11, pp. 1639-1654

2014

  1. Dual specificity phosphatase 1 expression inversely correlates with NF-κB activity and expression in prostate cancer and promotes apoptosis through a p38 MAPK dependent mechanism

    Molecular Oncology, Vol. 8, Núm. 1, pp. 27-38

2008

  1. Thyroid hormone-mediated activation of the ERK/dual specificity phosphatase 1 pathway augments the apoptosis of GH4C1 cells by down-regulating nuclear factor-κB activity

    Molecular Endocrinology, Vol. 22, Núm. 11, pp. 2466-2480

2006

  1. Low cell cholesterol levels increase NFκB activity through a p38 MAPK-dependent mechanism

    Cellular Signalling, Vol. 18, Núm. 12, pp. 2292-2301

  2. RhoA and p38 MAPK mediate apoptosis induced by cellular cholesterol depletion

    Apoptosis, Vol. 11, Núm. 7, pp. 1161-1173

1997

  1. Effect of mevalonate availability on the association of G-protein α-subunits with the plasma membrane in GH4C1 cells

    FEBS Letters, Vol. 401, Núm. 1, pp. 68-72

  2. Lovastatin decreases prolactin and growth hormone gene expression in GH4C1 cells through a cAMP dependent mechanism

    Molecular and Cellular Endocrinology, Vol. 130, Núm. 1-2, pp. 93-100

1995

  1. Effects of lovastatin on adenylyl cyclase activity and G proteins in GH4C1 cells

    FEBS Letters, Vol. 361, Núm. 1, pp. 46-50

1993

  1. Cholesterol cell content affects prolactin but not growth hormone release in GH4c1 cells

    Endocrinology, Vol. 132, Núm. 4, pp. 1701-1706