Guillermo
Bodega Magro
Catedrático/a de Universidad
Universidad Complutense de Madrid
Madrid, EspañaPublicaciones en colaboración con investigadores/as de Universidad Complutense de Madrid (52)
2023
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New mechanisms involved in the development of cardiovascular disease in chronic kidney disease
Nefrologia, Vol. 43, Núm. 1, pp. 63-80
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Plasma glycocalyx pattern: a mirror of endothelial damage in chronic kidney disease
Clinical Kidney Journal, Vol. 16, Núm. 8, pp. 1278-1287
2021
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A high magnesium concentration in citrate dialysate prevents oxidative stress and damage in human monocytes in vitro
Clinical Kidney Journal, Vol. 14, Núm. 5, pp. 1403-1411
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Premature aging in chronic kidney disease: The outcome of persistent inflammation beyond the bounds
International Journal of Environmental Research and Public Health, Vol. 18, Núm. 15
2020
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Hypoxia-Inducible Factor-1α: The Master Regulator of Endothelial Cell Senescence in Vascular Aging
Cells, Vol. 9, Núm. 1
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Increasing the magnesium concentration in various dialysate solutions differentially modulates oxidative stress in a human monocyte cell line
Antioxidants, Vol. 9, Núm. 4
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Mechanisms of Cardiovascular Disorders in Patients With Chronic Kidney Disease: A Process Related to Accelerated Senescence
Frontiers in Cell and Developmental Biology, Vol. 8
2019
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MicroRNA-126 regulates Hypoxia-Inducible Factor-1α which inhibited migration, proliferation, and angiogenesis in replicative endothelial senescence
Scientific Reports, Vol. 9, Núm. 1
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Microvesicles: ROS scavengers and ROS producers
Journal of Extracellular Vesicles, Vol. 8, Núm. 1
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Tendothelial extracellular vesicles produced by senescent cells: Pathophysiological role in the cardiovascular disease associated with all types of diabetes mellitus
Current Vascular Pharmacology, Vol. 17, Núm. 5, pp. 447-454
2018
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Senescent microvesicles: A novel advance in molecular mechanisms of atherosclerotic calcification
International Journal of Molecular Sciences, Vol. 19, Núm. 7
2017
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Microvesicles from the plasma of elderly subjects and from senescent endothelial cells promote vascular calcification
Aging, Vol. 9, Núm. 3, pp. 778-789
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Reduced TH expression and α-synuclein accumulation contribute towards nigrostriatal dysfunction in experimental hepatic encephalopathy
Restorative Neurology and Neuroscience, Vol. 35, Núm. 5, pp. 469-481
2012
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Ammonia induces aquaporin-4 rearrangement in the plasma membrane of cultured astrocytes
Neurochemistry International, Vol. 61, Núm. 8, pp. 1314-1324
2010
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Upregulation of α-synuclein expression in the rat cerebellum in experimental hepatic encephalopathy
Neuropathology and Applied Neurobiology, Vol. 36, Núm. 5, pp. 422-435
2009
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Astrocytes express Mxi2, a splice isoform of p38MAPK
Journal of Molecular Histology, Vol. 40, Núm. 5-6, pp. 325-329
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Induction of NOS and nitrotyrosine expression in the rat striatum following experimental hepatic encephalopathy
Metabolic Brain Disease, Vol. 24, Núm. 3, pp. 395-408
2007
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Effect of ammonia on ciliary neurotrophic factor mRNA and protein expression and its upstream signalling pathway in cultured rat astroglial cells: Possible implication of c-fos, Sp1 and p38MAPK
Neuropathology and Applied Neurobiology, Vol. 33, Núm. 4, pp. 420-430
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Effect of ammonia, glutamine, and serum on calcineurin, p38MAPK-diP, GADD153/CHOP10, and CNTF in primary rat astrocyte cultures
Brain Research, Vol. 1175, Núm. 1, pp. 126-133
2006
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Possible implication of ciliary neurotrophic factor (CNTF) and β-synuclein in the ammonia effect on cultured rat astroglial cells: A study using DNA and protein microarrays
Neurochemistry International, Vol. 48, Núm. 8, pp. 729-738