Publicaciones (74) Publicaciones de Francisco Javier de Lucio Cazaña

2024

  1. Prostaglandin Transporter and Dipeptidyl Peptidase-4 as New Pharmacological Targets in the Prevention of Acute Kidney Injury in Diabetes: An In Vitro Study

    International Journal of Molecular Sciences, Vol. 25, Núm. 6

2023

  1. Exploring the Metabolic Differences between Cisplatin- and UV Light-Induced Apoptotic Bodies in HK-2 Cells by an Untargeted Metabolomics Approach

    International Journal of Molecular Sciences, Vol. 24, Núm. 8

  2. Prostaglandin transporter PGT as a new pharmacological target in the prevention of inflammatory cytokine-induced injury in renal proximal tubular HK-2 cells

    Life Sciences, Vol. 313

2021

  1. Comprehensive metabolomic study of the response of HK-2 cells to hyperglycemic hypoxic diabetic-like milieu

    Scientific Reports, Vol. 11, Núm. 1

  2. Exploratory metabolomic analysis based on reversed‐phase liquid chromatography–mass spectrometry to study an in vitro model of hypoxia‐induced metabolic alterations in hk‐2 cells

    International Journal of Molecular Sciences, Vol. 22, Núm. 14

  3. Intracellular prostaglandin E2 contributes to hypoxia-induced proximal tubular cell death

    Scientific Reports, Vol. 11, Núm. 1

  4. Mitochondria selective trackers for long-term imaging based on readily accessible neutral BODIPYs

    Chemical Communications, Vol. 57, Núm. 43, pp. 5318-5321

2020

  1. A non-targeted capillary electrophoresis-mass spectrometry strategy to study metabolic differences in an in vitro model of high-glucose induced changes in human proximal tubular HK-2 cells

    Molecules, Vol. 25, Núm. 3

  2. Author Correction: Mechanism and Consequences of The Impaired Hif-1α Response to Hypoxia in Human Proximal Tubular HK-2 Cells Exposed to High Glucose (Scientific Reports, (2019), 9, 1, (15868), 10.1038/s41598-019-52310-6)

    Scientific Reports

  3. Retinoic acid receptor-beta prevents cisplatin-induced proximal tubular cell death

    Biochimica et Biophysica Acta - Molecular Basis of Disease, Vol. 1866, Núm. 7

  4. Time-series proteomic study of the response of HK-2 cells to hyperglycemic, hypoxic diabetic-like milieu

    PLoS ONE, Vol. 15, Núm. 6

2019

  1. PROSTAGLANDIN E 2 stimulates cancer-related phenotypes in prostate cancer PC3 cells through cyclooxygenase-2

    Journal of Cellular Physiology, Vol. 234, Núm. 5, pp. 7548-7559

  2. An untargeted metabolomic strategy based on liquid chromatography-mass spectrometry to study high glucose-induced changes in HK-2 cells

    Journal of Chromatography A, Vol. 1596, pp. 124-133

  3. Apoptosis and cell proliferation in proximal tubular cells exposed to apoptotic bodies. Novel pathophysiological implications in cisplatin-induced renal injury

    Biochimica et Biophysica Acta - Molecular Basis of Disease, Vol. 1865, Núm. 9, pp. 2504-2515

  4. Mechanism and Consequences of The Impaired Hif-1α Response to Hypoxia in Human Proximal Tubular HK-2 Cells Exposed to High Glucose

    Scientific Reports, Vol. 9, Núm. 1

  5. Papel renoprotector de DIDs en la citotoxicidad inducida por cisplatino

    Séptimas Jornadas de Jóvenes Investigadores de la Universidad de Alcalá: Ciencias e Ingenierías

2018

  1. Intracrine prostaglandin E2 pro-tumoral actions in prostate epithelial cells originate from non-canonical pathways

    Journal of Cellular Physiology, Vol. 233, Núm. 4, pp. 3590-3602

2017

  1. Aligned copper nanowires as a cut-and-paste exclusive electrochemical transducer for free-enzyme highly selective quantification of intracellular hydrogen peroxide in cisplatin-treated cells

    Biosensors and Bioelectronics, Vol. 96, pp. 146-151

2016

  1. Efecto de las microvesículas secretadas por células tumorales de próstata sobre células de próstata no tumorales

    Quintas Jornadas de Jóvenes Investigadores de la Universidad de Alcalá: Ciencias e Ingenierías

  2. Intracellular prostaglandin E2 mediates cisplatin-induced proximal tubular cell death

    Biochimica et Biophysica Acta - Molecular Cell Research, Vol. 1863, Núm. 2, pp. 293-302