José
Palacios Calvo
Profesor/a Titular Universidad
Centro Nacional de Investigaciones Oncológicas
Madrid, EspañaPublicaciones en colaboración con investigadores/as de Centro Nacional de Investigaciones Oncológicas (85)
2023
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CCNE1 and survival of patients with tubo-ovarian high-grade serous carcinoma: An Ovarian Tumor Tissue Analysis consortium study
Cancer, Vol. 129, Núm. 5, pp. 697-713
2021
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LungBEAM: A prospective multicenter study to monitor stage IV NSCLC patients with EGFR mutations using BEAMing technology
Cancer Medicine, Vol. 10, Núm. 17, pp. 5878-5888
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Tumor mutational burden assessment in non-small-cell lung cancer samples: Results from the TMB 2 harmonization project comparing three NGS panels
Journal for ImmunoTherapy of Cancer, Vol. 9, Núm. 5
2020
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Development and validation of the gene expression predictor of high-grade serous ovarian carcinoma molecular SubTYPE (PrOTYPE)
Clinical Cancer Research, Vol. 26, Núm. 20, pp. 5411-5423
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Mutational Screening of BRCA1/2 Genes as a Predictive Factor for Therapeutic Response in Epithelial Ovarian Cancer: A Consensus Guide from the Spanish Society of Pathology (SEAP-IAP) and the Spanish Society of Human Genetics (AEGH)
Virchows Archiv, Vol. 476, Núm. 2, pp. 195-207
2018
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Association of p16 expression with prognosis varies across ovarian carcinoma histotypes: an Ovarian Tumor Tissue Analysis consortium study
The journal of pathology. Clinical research, Vol. 4, Núm. 4, pp. 250-261
2017
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18F-fluoromisonidazole PET and activity of neoadjuvant nintedanib in early HER2-negative breast cancer: A window-of-opportunity randomized trial
Clinical Cancer Research, Vol. 23, Núm. 6, pp. 1432-1441
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Characterisation of the novel deleterious RAD51C p.Arg312Trp variant and prioritisation criteria for functional analysis of RAD51C missense changes
British Journal of Cancer, Vol. 117, Núm. 7, pp. 1048-1062
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Dose-Response Association of CD8+ Tumor-Infiltrating Lymphocytes and Survival Time in High-Grade Serous Ovarian Cancer
JAMA oncology, Vol. 3, Núm. 12, pp. e173290
2015
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Exome sequencing identifies ATP4A gene as responsible of an atypical familial type I gastricneuroendocrine tumour
Human Molecular Genetics, Vol. 24, Núm. 10, pp. 2914-2922
2014
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VGLL1 expression is associated with a triple-negative basal-like phenotype in breast cancer
Endocrine-Related Cancer, Vol. 21, Núm. 4, pp. 587-599
2013
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Analysis of DNA repair-related genes in breast cancer reveals CUL4A ubiquitin ligase as a novel biomarker of trabectedin response
Molecular Cancer Therapeutics, Vol. 12, Núm. 4, pp. 530-541
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DNA copy number profiling reveals extensive genomic loss in hereditary BRCA1 and BRCA2 ovarian carcinomas
British Journal of Cancer, Vol. 108, Núm. 8, pp. 1732-1742
2012
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A new multidisciplinary Spanish Working Group on Cancer Biomarkers: Presentation and aims
Clinical and Translational Oncology
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Classical markers like ER and Ki-67, but also survivin and pERK, could be involved in the pathological response to gemcitabine, adriamycin and paclitaxel (GAT) in locally advanced breast cancer patients: Results from the GEICAM/2002-01 phase II study
Clinical and Translational Oncology, Vol. 14, Núm. 6, pp. 430-436
2011
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Consensus of the Spanish Society of Medical Oncology (SEOM) and Spanish Society of Pathology (SEAP) for HER2 testing in gastric carcinoma
Clinical and Translational Oncology, Vol. 13, Núm. 9, pp. 636-651
2010
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An integrative genomic and transcriptomic analysis reveals molecular pathways and networks regulated by copy number aberrations in basal-like, HER2 and luminal cancers
Breast Cancer Research and Treatment, Vol. 121, Núm. 3, pp. 575-589
2009
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Comprehensive characterization of the DNA amplification at 13q34 in human breast cancer reveals TFDP1 and CUL4A as likely candidate target genes
Breast Cancer Research, Vol. 11, Núm. 6
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Functional characterization of E- and P-cadherin in invasive breast cancer cells
BMC Cancer, Vol. 9
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Loss of I6q in high grade breast cancer is associated with estrogen receptor status: Evidence for progression in tumors with a luminal phenotype?
Genes Chromosomes and Cancer, Vol. 48, Núm. 4, pp. 351-365