Publicaciones en colaboración con investigadores/as de Hospital Universitario Príncipe de Asturias (132)

2021

  1. Effective glutamate clearance from the systemic circulation by hemodialysis: Potential relevance for cerebral ischemia management

    Artificial Organs, Vol. 45, Núm. 10, pp. 1183-1188

  2. Epicardial fat tissue, coronary arterial calcification and mortality in patients with advanced chronic kidney disease and hemodialysis

    Nefrologia, Vol. 41, Núm. 2, pp. 174-181

  3. Evaluating service-oriented and microservice architecture patterns to deploy ehealth applications in cloud computing environment

    Applied Sciences (Switzerland), Vol. 11, Núm. 10

  4. Impact of in-hospital discontinuation with angiotensin receptor blockers or converting enzyme inhibitors on mortality of COVID-19 patients: a retrospective cohort study

    BMC Medicine, Vol. 19, Núm. 1

  5. Interplay between extracellular matrix components and cellular and molecular mechanisms in kidney fibrosis

    Clinical Science, Vol. 135, Núm. 16, pp. 1999-2029

  6. Kidney and hypertension in older adults

    Medicina Clinica, Vol. 157, Núm. 4, pp. 178-184

  7. La interacción de los arquitectos con los profesionales de la medicina: el ejemplo de la construcción de una unidad de diálisis

    Habitar los hospitales: el bienestar más allá del confort: nuevas tendencias en arquitectura sanitaria (Editorial Universidad de Alcalá), pp. 150-157

  8. Medical prognosis of infectious diseases in nursing homes by applying machine learning on clinical data collected in cloud microservices

    International Journal of Environmental Research and Public Health, Vol. 18, Núm. 24

  9. Predicting the appearance of hypotension during hemodialysis sessions using machine learning classifiers

    International Journal of Environmental Research and Public Health, Vol. 18, Núm. 5, pp. 1-17

  10. The program of renal fibrogenesis is controlled by microRNAs regulating oxidative metabolism

    Redox Biology, Vol. 40

  11. Uraemic toxins impair skeletal muscle regeneration by inhibiting myoblast proliferation, reducing myogenic differentiation, and promoting muscular fibrosis

    Scientific Reports, Vol. 11, Núm. 1