Publicacións nas que colabora con Diego María Rodríguez Puyol (37)

2023

  1. Effects of a Losartan-Antioxidant Hybrid (GGN1231) on Vascular and Cardiac Health in an Experimental Model of Chronic Renal Failure

    Nutrients, Vol. 15, Núm. 8

2022

  1. RICORS2040: The need for collaborative research in chronic kidney disease

    Clinical Kidney Journal, Vol. 15, Núm. 3, pp. 372-387

2021

  1. Uraemic toxins impair skeletal muscle regeneration by inhibiting myoblast proliferation, reducing myogenic differentiation, and promoting muscular fibrosis

    Scientific Reports, Vol. 11, Núm. 1

2020

  1. Endothelin-1 induces cellular senescence and fibrosis in cultured myoblasts. A potential mechanism of aging-related sarcopenia

    Aging, Vol. 12, Núm. 12, pp. 11200-11223

2018

  1. Hyperphosphatemia promotes senescence of myoblasts by impairing autophagy through Ilk overexpression, a possible mechanism involved in sarcopenia

    Aging and Disease, Vol. 9, Núm. 5, pp. 769-784

  2. Impaired erythropoietin synthesis in chronic kidney disease is caused by alterations in extracellular matrix composition

    Journal of Cellular and Molecular Medicine, Vol. 22, Núm. 1, pp. 302-314

2017

  1. Hyperphosphatemia induces senescence in human endothelial cells by increasing endothelin-1 production

    Aging Cell, Vol. 16, Núm. 6, pp. 1300-1312

2016

  1. Erratum: Glucose Oxidase Induces Cellular Senescence in Immortal Renal Cells through ILK by Downregulating Klotho Gene Expression(Oxidative Medicine and Cellular Longevity (2015) 2015 (13))

    Oxidative Medicine and Cellular Longevity

2015

  1. Glucose Oxidase Induces Cellular Senescence in Immortal Renal Cells through ILK by Downregulating Klotho Gene Expression

    Oxidative Medicine and Cellular Longevity, Vol. 2015

  2. Hyperphosphatemia induces cellular senescence in human aorta smooth muscle cells through integrin linked kinase (ILK) up-regulation

    Mechanisms of Ageing and Development, Vol. 152, pp. 43-55

  3. Renal integrin-linked kinase depletion induces kidney cGMP-axis upregulation: Consequences on basal and acutely damaged renal function

    Molecular Medicine, Vol. 21, pp. 873-885

2014

  1. Hyperosmolarity induced by high glucose promotes senescence in human glomerular mesangial cells

    International Journal of Biochemistry and Cell Biology, Vol. 54, pp. 98-110

2013

  1. Amadori products promote cellular senescence activating insulin-like growth factor-1 receptor and down-regulating the antioxidant enzyme catalase

    International Journal of Biochemistry and Cell Biology, Vol. 45, Núm. 7, pp. 1255-1264

  2. Ilk conditional deletion in adult animals increases cyclic GMP-dependent vasorelaxation

    Cardiovascular Research, Vol. 99, Núm. 3, pp. 535-544

2012

  1. Integrin-linked kinase (ILK) modulates wound healing through regulation of hepatocyte growth factor (HGF)

    Experimental Cell Research, Vol. 318, Núm. 19, pp. 2470-2481

2009

  1. Tirofiban increases soluble guanylate cyclase in rat vascular walls: Pharmacological and pathophysiological consequences

    Cardiovascular Research, Vol. 82, Núm. 1, pp. 125-132

2008

  1. Telomerase deficiency promotes oxidative stress by reducing catalase activity

    Free Radical Biology and Medicine, Vol. 45, Núm. 9, pp. 1243-1251

2007

  1. Role of activator protein-1 on the effect of arginine-glycine-aspartic acid containing peptides on transforming growth factor-β1 promoter activity

    International Journal of Biochemistry and Cell Biology, Vol. 39, Núm. 1, pp. 133-145

2006

  1. Arg-Gly-Asp (RGD)-containing peptides increase soluble guanylate cyclase in contractile cells

    Cardiovascular Research, Vol. 69, Núm. 2, pp. 359-369

  2. Mice deficient in telomerase activity develop hypertension because of an excess of endothelin production

    Circulation, Vol. 114, Núm. 4, pp. 309-317