Publikationen (59) Publikationen von María Piedad Ruiz Torres

2024

  1. Soluble Klotho, a Potential Biomarker of Chronic Kidney Disease–Mineral Bone Disorders Involved in Healthy Ageing: Lights and Shadows

    International Journal of Molecular Sciences, Vol. 25, Núm. 3

2023

  1. Dietary phosphate restriction prevents the appearance of sarcopenia signs in old mice

    Journal of Cachexia, Sarcopenia and Muscle, Vol. 14, Núm. 2, pp. 1060-1074

  2. Effects of a Losartan-Antioxidant Hybrid (GGN1231) on Vascular and Cardiac Health in an Experimental Model of Chronic Renal Failure

    Nutrients, Vol. 15, Núm. 8

2022

  1. RICORS2040: The need for collaborative research in chronic kidney disease

    Clinical Kidney Journal, Vol. 15, Núm. 3, pp. 372-387

  2. Vitamin D Treatment Prevents Uremia-Induced Reductions in Aortic microRNA-145 Attenuating Osteogenic Differentiation despite Hyperphosphatemia

    Nutrients, Vol. 14, Núm. 13

2021

  1. Aging-related hyperphosphatemia impairs myogenic differentiation and enhances fibrosis in skeletal muscle

    Journal of Cachexia, Sarcopenia and Muscle, Vol. 12, Núm. 5, pp. 1266-1279

  2. Contribution of phosphorus and PTH to the development of cardiac hypertrophy and fibrosis in an experimental model of chronic renal failure

    Nefrologia, Vol. 41, Núm. 6, pp. 640-651

  3. Effects of calcitriol and paricalcitol on renal fibrosis in CKD

    Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association, Vol. 36, Núm. 5, pp. 793-803

  4. Hyperphosphatemia-induced oxidant/antioxidant imbalance impairs vascular relaxation and induces inflammation and fibrosis in old mice

    Antioxidants, Vol. 10, Núm. 8

  5. Role of the RANK/RANKL/OPG and Wnt/β-Catenin Systems in CKD Bone and Cardiovascular Disorders

    Calcified Tissue International, Vol. 108, Núm. 4, pp. 439-451

  6. Uraemic toxins impair skeletal muscle regeneration by inhibiting myoblast proliferation, reducing myogenic differentiation, and promoting muscular fibrosis

    Scientific Reports, Vol. 11, Núm. 1

2020

  1. Endothelin-1 induces cellular senescence and fibrosis in cultured myoblasts. A potential mechanism of aging-related sarcopenia

    Aging, Vol. 12, Núm. 12, pp. 11200-11223

2018

  1. Hyperphosphatemia promotes senescence of myoblasts by impairing autophagy through Ilk overexpression, a possible mechanism involved in sarcopenia

    Aging and Disease, Vol. 9, Núm. 5, pp. 769-784

  2. Impaired erythropoietin synthesis in chronic kidney disease is caused by alterations in extracellular matrix composition

    Journal of Cellular and Molecular Medicine, Vol. 22, Núm. 1, pp. 302-314

2017

  1. Effects of the catalase antioxidant enzyme in vascular calcification and bone demineralization

    Revista de Osteoporosis y Metabolismo Mineral, Vol. 9, Núm. 1, pp. 13-19

  2. Hyperphosphatemia induces senescence in human endothelial cells by increasing endothelin-1 production

    Aging Cell, Vol. 16, Núm. 6, pp. 1300-1312

  3. Integrin-linked kinase: A new actor in the ageing process?

    Experimental Gerontology

  4. Microvesicles from the plasma of elderly subjects and from senescent endothelial cells promote vascular calcification

    Aging, Vol. 9, Núm. 3, pp. 778-789

2016

  1. Erratum: Glucose Oxidase Induces Cellular Senescence in Immortal Renal Cells through ILK by Downregulating Klotho Gene Expression(Oxidative Medicine and Cellular Longevity (2015) 2015 (13))

    Oxidative Medicine and Cellular Longevity

  2. Lamin A is involved in the development of vascular calcification induced by chronic kidney failure and phosphorus load

    Bone, Vol. 84, pp. 160-168