Publications by the researcher in collaboration with Coral Bravo Arribas (24)

2024

  1. Exacerbated Activation of the NLRP3 Inflammasome in the Placentas from Women Who Developed Chronic Venous Disease during Pregnancy

    International Journal of Molecular Sciences, Vol. 25, Núm. 10

  2. Exploring the Importance of Differential Expression of Autophagy Markers in Term Placentas from Late-Onset Preeclamptic Pregnancies

    International Journal of Molecular Sciences, Vol. 25, Núm. 4

  3. Histopathological Clues of Enhanced Inflammation in the Placental Tissue of Women with Chronic Venous Disease in Lower Limbs during Pregnancy

    Journal of Personalized Medicine, Vol. 14, Núm. 1

  4. Oxidative Stress, Lipid Peroxidation and Ferroptosis Are Major Pathophysiological Signatures in the Placental Tissue of Women with Late-Onset Preeclampsia

    Antioxidants, Vol. 13, Núm. 5

  5. Sudden Infant Death Syndrome (SIDS): State of the Art and Future Directions

    International Journal of Medical Sciences, Vol. 21, Núm. 5, pp. 848-861

2023

  1. Assessment of Tissue Expression of the Oxytocin–Vasopressin Pathway in the Placenta of Women with a First-Episode Psychosis during Pregnancy

    International Journal of Molecular Sciences, Vol. 24, Núm. 12

  2. Chronic Venous Disease during Pregnancy Is Related to Inflammation of the Umbilical Cord: Role of Allograft Inflammatory Factor 1 (AIF-1) and Interleukins 10 (IL-10), IL-12 and IL-18

    Journal of Personalized Medicine, Vol. 13, Núm. 6

  3. Evidence of Increased Oxidative Stress in the Placental Tissue of Women Who Suffered an Episode of Psychosis during Pregnancy

    Antioxidants, Vol. 12, Núm. 1

  4. Exploring the Role of Mediterranean and Westernized Diets and Their Main Nutrients in the Modulation of Oxidative Stress in the Placenta: A Narrative Review

    Antioxidants, Vol. 12, Núm. 11

  5. Increased Expression of Tetraspanins, ALIX and HSP-70 in the Placenta Tissue of Women with Chronic Venous Disease during Pregnancy

    International Journal of Medical Sciences, Vol. 20, Núm. 13, pp. 1744-1754

  6. Placentas from Women with Late-Onset Preeclampsia Exhibit Increased Expression of the NLRP3 Inflammasome Machinery

    Biomolecules, Vol. 13, Núm. 11

  7. The Placentas of Women Who Suffer an Episode of Psychosis during Pregnancy Have Increased Lipid Peroxidation with Evidence of Ferroptosis

    Biomolecules, Vol. 13, Núm. 1

  8. Women with psychotic episodes during pregnancy show increased markers of placental damage with Tenney-Parker changes

    Histology and Histopathology, Vol. 38, Núm. 10, pp. 1109-1118

2022

  1. Chronic Venous Disease in Pregnant Women Causes an Increase in ILK in the Placental Villi Associated with a Decrease in E-Cadherin

    Journal of Personalized Medicine, Vol. 12, Núm. 2

  2. Considering the Effects and Maternofoetal Implications of Vascular Disorders and the Umbilical Cord

    Medicina (Lithuania), Vol. 58, Núm. 12

  3. Overexpression of glycolysis markers in placental tissue of pregnant women with chronic venous disease: a histological study

    International journal of medical sciences, Vol. 19, Núm. 1, pp. 186-194

  4. Patients with Invasive Lobular Carcinoma Show a Significant Increase in IRS-4 Expression Compared to Infiltrative Ductal Carcinoma—A Histopathological Study

    Medicina (Lithuania), Vol. 58, Núm. 6

  5. The Pivotal Role of the Placenta in Normal and Pathological Pregnancies: A Focus on Preeclampsia, Fetal Growth Restriction, and Maternal Chronic Venous Disease

    Cells, Vol. 11, Núm. 3

2021

  1. Abnormal proinflammatory and stressor environmental with increased the regulatory cellular igf-1/papp-a/ stc and wnt-1/β-catenin canonical pathway in placenta of women with chronic venous disease during pregnancy

    International Journal of Medical Sciences, Vol. 18, Núm. 13, pp. 2814-2827

  2. An increase in elastogenic components in the placental villi of women with chronic venous disease during pregnancy is associated with decreased EGFL7 expression level

    Molecular Medicine Reports, Vol. 24, Núm. 2