Publicaciones en las que colabora con José Luís Zamorano Gómez (21)

2024

  1. NOS3 prevents MMP-9, and MMP-13 induced extracellular matrix proteolytic degradation through specific microRNA-targeted expression of extracellular matrix metalloproteinase inducer in hypertension-related atherosclerosis

    Journal of Hypertension, Vol. 42, Núm. 4, pp. 685-693

  2. Pupil reflex as a marker of activity and prognosis in heart failure: a longitudinal and prospective study

    ESC Heart Failure, Vol. 11, Núm. 4, pp. 1947-1954

  3. Replicative Endothelial Cell Senescence May Lead to Endothelial Dysfunction by Increasing the BH2/BH4 Ratio Induced by Oxidative Stress, Reducing BH4 Availability, and Decreasing the Expression of eNOS

    International Journal of Molecular Sciences, Vol. 25, Núm. 18

2023

  1. Endothelial ILK induces cardioprotection by preventing coronary microvascular dysfunction and endothelial-to-mesenchymal transition

    Basic research in cardiology, Vol. 118, Núm. 1, pp. 28

2022

  1. Aging-related ILK levels are associated with calcified aortic valve and circulating miR 199-3p levels

    Revista Espanola de Cardiologia, Vol. 75, Núm. 1, pp. 88-91

  2. Corrigendum: Preclinical models of congestive heart failure, advantages and limitations for application in clinical practice (Front. Physiol., (2022), 13, 850301, 10.3389/fphys.2022.850301)

    Frontiers in Physiology

  3. Integrin-Linked Kinase Expression in Human Valve Endothelial Cells Plays a Protective Role in Calcific Aortic Valve Disease

    Antioxidants, Vol. 11, Núm. 9

  4. NIL10: A New IL10-Receptor Binding Nanoparticle That Induces Cardiac Protection in Mice and Pigs Subjected to Acute Myocardial Infarction through STAT3/NF-κB Activation

    Pharmaceutics, Vol. 14, Núm. 10

  5. Preclinical models of congestive heart failure, advantages, and limitations for application in clinical practice

    Frontiers in Physiology, Vol. 13

  6. Theranostic Contribution of Extracellular Matrix Metalloprotease Inducer-Paramagnetic Nanoparticles Against Acute Myocardial Infarction in a Pig Model of Coronary Ischemia-Reperfusion

    Circulation. Cardiovascular imaging, Vol. 15, Núm. 6, pp. e013379

2021

  1. Ivabradine induces cardiac protection against myocardial infarction by preventing cyclophilin‐a secretion in pigs under coronary ischemia/reperfusion

    International Journal of Molecular Sciences, Vol. 22, Núm. 6, pp. 1-13

  2. Ivabradine induces cardiac protection by preventing cardiogenic shock-induced extracellular matrix degradation

    Revista Espanola de Cardiologia, Vol. 74, Núm. 12, pp. 1063-1072

  3. Safety of low-dose oral minoxidil for hair loss: A multicenter study of 1404 patients

    Journal of the American Academy of Dermatology, Vol. 84, Núm. 6, pp. 1644-1651

2020

  1. Ivabradine in acute heart failure: Effects on heart rate and hemodynamic parameters in a randomized and controlled swine trial

    Cardiology Journal, Vol. 27, Núm. 1, pp. 62-71

  2. Ivabradine-stimulated microvesicle release induces cardiac protection against acute myocardial infarction

    International Journal of Molecular Sciences, Vol. 21, Núm. 18, pp. 1-12

2019

  1. Differential expression of circulating miRNAs as a novel tool to assess BAG3-associated familial dilated cardiomyopathy

    Bioscience Reports, Vol. 39, Núm. 3

  2. Nanotechnology Applied to Preserve Extracelular Matrix as Teranostic Tool in Acute Myocardial Infarction

    Revista Espanola de Cardiologia, Vol. 72, Núm. 2, pp. 171-174

2018

  1. Non-invasive detection of extracellular matrix metalloproteinase inducer EMMPRIN, a new therapeutic target against atherosclerosis, inhibited by endothelial nitric oxide

    International Journal of Molecular Sciences, Vol. 19, Núm. 10

2016

  1. EMMPRIN-Targeted magnetic nanoparticles for in vivo visualization and regression of acute myocardial infarction

    Theranostics, Vol. 6, Núm. 4, pp. 545-557

  2. Nitric oxide induces cardiac protection by preventing extracellular matrix degradation through the complex caveolin-3/emmprin in cardiac myocytes

    PLoS ONE, Vol. 11, Núm. 9